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RV pacing dyssynchrony on echocardiography
Similar to LBBB: abnormal activation from the RV apex, with a pacing spike before a broad QRS on the ECG. Thickening is preserved.
Watch it in the simulator →Practise random casesWhere to look
Apical 4-chamber and PSAX; the ECG strip shows a pacing spike before a broad QRS.
Typical wall-motion pattern
Example: RV apical pacing
All 17 segments thicken normally.
EF in this example: 42% (Impaired (36–49%)).
1 Normal2 Hypokinetic3 Akinetic4 Dyskinetic5 Aneurysmal
Ejection fraction
Across 24 simulated cases of this pattern the true EF ranged from 39% to 46% (median 41%, Impaired (36–49%)). Every case in the simulator is different, so practise until your visual estimate lands in the right 5% range.
Teaching points
- RV pacing: activation starts at the RV apex, so the apex and septum contract first (early septal flick) and the basal lateral wall last, with apical rocking. Look for the pacing lead in the RV and the pacing spike on the ECG. Thickening is preserved.
Common pitfalls
As with LBBB, abnormal motion with preserved thickening is not an RWMA.
Try it now: open the RV pacing dyssynchrony example with the segment overlay, then test yourself in Practice where cases are random and graded against the exact answer.
Other patterns
- Normal LV function on echocardiography
- Hyperdynamic left ventricle on echo
- Regional wall motion abnormalities and coronary territories on echo
- Left ventricular aneurysm on echocardiography
- LV thrombus on echocardiography
- Dilated cardiomyopathy and global hypokinesia on echo
- Takotsubo cardiomyopathy on echocardiography
- LBBB on echo: septal flash, apical rocking and dyssynchrony
- Paradoxical septal motion after cardiac surgery
- RV pressure overload on echo: systolic D-shaped septum
- RV volume overload on echo: diastolic D-shape and paradoxical septal motion
- Combined RV pressure and volume overload on echo: D-shape in systole and diastole
- Concentric LV hypertrophy on echocardiography
- Hypertrophic cardiomyopathy on echo: asymmetric septal hypertrophy and SAM
- LV non-compaction on echocardiography